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عمومی::
ادم مغزی، ادم مغزی
□ The resolution of cerebral edema depends on hydrostatic and osmolar forces applied to the blood-brain barrier.
As a reduction of plasma osmolality of 4 to 5 mOsm/kg increases cerebral edema, hypotonic solutions must not be used in the neurosurgical patient.
Sub- sequent effects on CBF, microcirculatory changes, cerebral edema, and sympathetic response can lead to both neuro- logic and systemic complications.150,151 Acute management of blood pressure and oxygenation are critical in this period along with the prevention of early complications such as rebleeding, acute hydrocephalus, and elevated ICPs.
Early management of the potential complications of AIS, including postinfarction cerebral edema, hemor- rhagic transformation, and reperfusion injury, are critical to improving patient outcomes.
Changes in the patient examination after AIS may indi- cate a hemorrhagic complication, cerebral hyperperfu- sion, or cerebral edema and should prompt an emergent noncontrast CT.،□ The resolution of cerebral edema depends on hydrostatic and osmolar forces applied to the blood-brain barrier.
As a reduction of plasma osmolality of 4 to 5 mOsm/kg increases cerebral edema, hypotonic solutions must not be used in the neurosurgical patient.
Sub- sequent effects on CBF, microcirculatory changes, cerebral edema, and sympathetic response can lead to both neuro- logic and systemic complications.150,151 Acute management of blood pressure and oxygenation are critical in this period along with the prevention of early complications such as rebleeding, acute hydrocephalus, and elevated ICPs.
Early management of the potential complications of AIS, including postinfarction cerebral edema, hemor- rhagic transformation, and reperfusion injury, are critical to improving patient outcomes.
Changes in the patient examination after AIS may indi- cate a hemorrhagic complication, cerebral hyperperfu- sion, or cerebral edema and should prompt an emergent noncontrast CT.
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